Defining the relationships between muscle injury and muscle repair is critical within the field of rehabilitation. Muscle is a highly adaptable tissue that responds to internal and external stimulus – whether to botulinum toxin injections or intervertebral disc degeneration, deletion of protein desmin or nephropathic cystinosis. Further comprehension of these relationships and responses may highlight opportunities to improving timing and types of treatment.
- Muscle wasting and adipose tissue browning in infantile nephropathic cystinosis.
- Dramatic changes in muscle contractile and structural properties after 2 botulinum toxin injections.
- Lmo7 is dispensable for skeletal muscle and cardiac function.
- Disruption of both nesprin 1 and desmin results in nuclear anchorage defects and fibrosis in skeletal muscle.
- Effect of supraspinatus tendon injury on supraspinatus and infraspinatus muscle passive tension and associated biochemistry.
- Muscle gene expression patterns in human rotator cuff pathology.
- Contribution of denervated muscle to contractures after neonatal brachial plexus injury: not just muscle fibrosis.
- Loss of FHL1 induces an age-dependent skeletal muscle myopathy associated with myofibrillar and intermyofibrillar disorganization in mice.